By Eric Guaguere, Pascal Prelaud, D.W. Scott, M. Craig

A pragmatic consultant to pussycat Dermatology

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7). It is assumed that there are also bits of retained adherent keratinocytes in this material, and it is generally understood that loose keratin under the skin is not welcomed by the innate immune system. When an epidermoid cyst ruptures under the skin, exposing released keratin, it is sterile but it causes a massively hot and tender inflammatory reaction that is often mistaken by the unaware for infection. The resolution of such lesions, brought about by simply removing the keratin (and the germina­ tive epithelium surrounding it), is both swift and impressive.

No sebaceous gland remains. has existed since the inaccurate assignment of responsibility to the “sweat glands” by Verneuil well over a century ago and specifically to the apocrine sweat glands by Brunsting in 1939 [13]. Note that there is no destruction or even inflammation directed at the apocrine glands themselves. 14). This disease was never a hidradenitis; it has always been a variant of acne. 13 The outline of the residual parts of the pilar unit is just barely visible. know what is happening in the top end of the follicle in sun-exposed FPSUs.

The males actually come out at night on the skin surface, mate, and then return to their pores. One wonders if the females also are night wanderers; otherwise, how would the infestation spread? The mites have been considered by many dermatologists over the years to be simply innocent bystanders, but there are recent clues that they are likely seriously involved in some, although perhaps not all, cases of acne rosacea [6]. One of the clues is from simply sampling the material in the little pustules that show up on patients’ faces.

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A Practical guide to Feline Dermatology by Eric Guaguere, Pascal Prelaud, D.W. Scott, M. Craig


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